Cardiology Research, ISSN 1923-2829 print, 1923-2837 online, Open Access
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Case Report

Volume 7, Number 5, October 2016, pages 178-180


Cardiac Amyloidosis Masked as Hypertrophic Cardiomyopathy: A Case Report

Figure

Figure 1.
Figure 1. Different LGE patterns between two CMRs. The upper panel showed mid-myocardial delayed enhancement in first CMR (October 2015), and the lower panel showed global subendocardial enhancement in second CMR (March 2016).

Table

Table 1. Characteristics of Examination Findings
 
20152016
IVS: interventricular septum; LVEF: left ventricular ejection fraction; CMR: cardiac magnetic resonance; LGE: late gadolinium enhancement.
ECGSinus rhythm
Normal QRS voltage
Q waves in the inferior leads and from V1 to V4
Sinus rhythm
Low QRS voltages
Q waves in the inferior leads and from V1 to V4
EchocardiographyIVS thickness: 1.6 cm
Normal LVEF of 61%
Dilated LA
Severe diastolic dysfunction
IVS thickness: 1.6 cm
Normal LVEF of 55%
Dilated LA
Severe diastolic dysfunction
CMRModerate hypertrophy without obstruction (IVS thickness 1.6 cm)
Normal size with mildly decreased cardiac function (LVEF 46%)
Pattern of LGE: faint mid-myocardial delayed enhancement at the infero-septum from base to mid-cavity
Mildly hypertrophy without obstruction (IVS thickness 1.2 cm)
Normal size with global hypokinesis (LVEF 41%)
Pattern of LGE: subendocardial enhancement
DiagnosisHypertrophic cardiomyopathyCardiac amyloidosis